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<br>The speed of fall of the glycogen was linear through the period of aglycemia. This instructed that if glycogen was unregulated prior to introducing aglycemia then the latency to CAP failure might be extended if glycogen was metabolized at the same steady price. Glycogen can be unregulated by bathing the nerve in supra-physiological concentrations of glucose, thus incubating the nerve in 30 mM glucose for two h increases the glycogen content material by an element of about two compared to baseline ranges, the place the nerve is incubated in 10 mM glucose for 2 h (Wender et al., 2000). Exposing the optic nerve to aglycemia after growing glycogen content did indeed lead to an increase in the latency to CAP failure. 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